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Feline hypoadrenocorticism
/content/chapter/10.22233/9781910443866.chap31
Feline hypoadrenocorticism
- Authors: Samuel Fowlie and Ian K. Ramsey
- From: BSAVA Manual of Canine and Feline Endocrinology
- Item: Chapter 31, pp 303 - 308
- DOI: 10.22233/9781910443866.31
- Copyright: © 2023 British Small Animal Veterinary Association
- Publication Date: August 2023
Abstract
Feline hypoadrenocorticism is a rare condition characterized by reduced secretion of glucocorticoids and mineralocorticoids. This chapter covers its prevalence and causes. Primary and secondary hypoadrenocorticism are discussed, as well as atypical cases. The diagnostic process, including clinical features, laboratory testing and imaging, is also covered. Treatment options, which include fluid therapy and glucocorticoid supplementation, are outlined. The chapter concludes with a discussion of the prognosis for cats with hypoadrenocorticism.
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Figures
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31.3
Causes of low sodium:potassium in 49 cats without hypoadrenocorticism. All 49 cats had hyperkalaemia; nine also had hyponatraemia. No cats with hypoadrenocorticism were identified while these cases were collected, emphasizing the rarity of feline hypoadrenocorticism as a cause of sodium and potassium abnormalities.
(Data from
Bell et al., 2005
) © 2023 British Small Animal Veterinary Association
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31.3
Causes of low sodium:potassium in 49 cats without hypoadrenocorticism. All 49 cats had hyperkalaemia; nine also had hyponatraemia. No cats with hypoadrenocorticism were identified while these cases were collected, emphasizing the rarity of feline hypoadrenocorticism as a cause of sodium and potassium abnormalities.
(Data from
Bell et al., 2005
)
/content/figure/10.22233/9781910443866.chap31.fig31_4
31.4
A thin (2.2kg) cat with history of waxing and waning lethargy, reduced appetite and soft faeces that was diagnosed with primary hypoadrenocorticism on the basis of hyponatraemia with normal potassium concentration, a failure of cortisol secretion to respond to ACTH stimulation and a greatly increased endogenous ACTH concentration (323 pmol/l; reference interval 8–39 pmol/l). © 2023 British Small Animal Veterinary Association
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10.22233/9781910443866/fig31_4.png
31.4
A thin (2.2kg) cat with history of waxing and waning lethargy, reduced appetite and soft faeces that was diagnosed with primary hypoadrenocorticism on the basis of hyponatraemia with normal potassium concentration, a failure of cortisol secretion to respond to ACTH stimulation and a greatly increased endogenous ACTH concentration (323 pmol/l; reference interval 8–39 pmol/l).